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Raf-1 activation disrupts its binding to keratins during cell stress
Nam On Ku
, Haian Fu, M. Bishr Omary
Interdisciplinary Program of Integrated OMICS for Biomedical Sciences
Research output
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Contribution to journal
›
Article
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peer-review
48
Citations (Scopus)
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Dive into the research topics of 'Raf-1 activation disrupts its binding to keratins during cell stress'. Together they form a unique fingerprint.
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Biochemistry, Genetics and Molecular Biology
Association
100%
Keratin
83%
Phosphorylation
33%
Raf Kinase
33%
Keratin 8
33%
Keratin 18
33%
Signal Transduction
16%
Cell Stress
16%
14-3-3 Protein
16%
Animal
16%
Cell Culture
16%
Hyperphosphorylation
16%
Keratin 14
16%
Neuroscience
Keratin
83%
Raf Kinase
50%
Phosphorylation
33%
Cytokeratin 8
33%
Cytokeratin 18
33%
Cell Signaling
16%
Cell Stress
16%
Cytokeratin 14
16%
Hyperphosphorylation
16%
14-3-3 Protein
16%
Pharmacology, Toxicology and Pharmaceutical Science
Keratin
83%
Raf Protein
50%
Cytokeratin 18
33%
Cytokeratin 8
33%
K 18
33%
Animal
16%
Toxin
16%
Protein 14 3 3
16%
Cytokeratin 14
16%